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Study of the mechanism of prostacyclin (PgI2) action of myocardial contractility

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dc.contributor.authorPavlovic, M. (35335027800)
dc.contributor.authorPetkovic, D. (7006263342)
dc.contributor.authorCvetkovic, M. (59022151300)
dc.contributor.authorZdjelar, K. (6505993671)
dc.contributor.authorStarcevic, V. (7005374307)
dc.contributor.authorBosnic, O. (6508388358)
dc.date.accessioned2025-07-02T12:58:23Z
dc.date.available2025-07-02T12:58:23Z
dc.date.issued1992
dc.description.abstractThe experiments done on the isolated right ventricle of rat hear suspended in the bath for isolated organs with oxygenated Tyrode's solution showed that PgI2 (2,3 x 10-6 mmol) produced its positive inotropic effect indirectly by promoting both beta-adrenoreceptors and calcium entry through slow calcium channels because this effect could be blocked by propranolol (2,3 x 10-2 mmol - beta adrenoreceptor blocker) or verapamil (4,3 x l0-3 mmol - slow calcium channel blocker).
dc.identifier.urihttps://doi.org/10.1007/978-3-0348-7262-1_23
dc.identifier.urihttps://www.scopus.com/inward/record.uri?eid=2-s2.0-0026597823&doi=10.1007%2f978-3-0348-7262-1_23&partnerID=40&md5=0c303fa4f9f3d60563b588f902f5aa63
dc.identifier.urihttps://remedy.med.bg.ac.rs/handle/123456789/14640
dc.titleStudy of the mechanism of prostacyclin (PgI2) action of myocardial contractility
dspace.entity.typePublication

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