Kostic, V.S. (35239923400)V.S. (35239923400)KosticDrulovic, B. (6508386868)B. (6508386868)DrulovicTodorovic, D. (57215783187)D. (57215783187)Todorovic2025-06-122025-06-121988https://www.scopus.com/inward/record.uri?eid=2-s2.0-0023907303&partnerID=40&md5=1e8c9d45a4b5433fdf1ba50dba76acadhttps://remedy.med.bg.ac.rs/handle/123456789/1980In 19 patients affected by Friedreich's ataxia, abnormalities of visual evoked potentials were recorded in 11 patients (58%). The most frequently encountered finding was a prolongation of the P 100 component latency (47%), although a reduction in the amplitude and an increase in the time interval between successive peaks was also observed. Pathological visual evoked potentials were also recorded in 18 clinically healthy members of 7 families of our patients (parents and siblings). Increased latency of the P 100 component was observed in 6 (33%).