Tanriverdi, F. (6602098737)F. (6602098737)TanriverdiAgha, A. (57238994100)A. (57238994100)AghaAimaretti, G. (7003308152)G. (7003308152)AimarettiCasanueva, F.F. (7103087629)F.F. (7103087629)CasanuevaKelestimur, F. (7005991966)F. (7005991966)KelestimurKlose, M. (56243195800)M. (56243195800)KloseMasel, B.E. (6603178453)B.E. (6603178453)MaselPereira, A.M. (7402230059)A.M. (7402230059)PereiraPopovic, V. (35451450900)V. (35451450900)PopovicSchneider, H.J. (25940508700)H.J. (25940508700)Schneider2025-06-122025-06-122011https://doi.org/10.3275/7805https://www.scopus.com/inward/record.uri?eid=2-s2.0-80054109571&doi=10.3275%2f7805&partnerID=40&md5=55abe4f47d34a4b283adf2dcabfb8975https://remedy.med.bg.ac.rs/handle/123456789/10012Traumatic brain injury (TBI)-induced hypopituitarism remains a relevant medical problem, because it may affect a significant proportion of the population. In the last decade important studies have been published investigating pituitary dysfunction after TBI. Recently, a group of experts gathered and revisited the topic of TBI-induced hypopitu-itarism. During the 2-day meeting, the main issues of this topic were presented and discussed, and current understanding and management of TBI-induced hypopituitarism are summarized here. ©2011, Editrice Kurtis.HypopituitarismTraumatic brain injury